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为了对人体心脏按压的压力传导机制作进一步研究，本文对7例危重病人（脑外伤4例，GCS均为3分，多发伤3例，平均ISS为22．3分）在高级生命支持下，心肺停止后进行常规抢救过程中，通过Swan-Ganz导管和有创股动脉压监测得到右房压（RAP），肺动脉压（PAP），股动脉压（FAP）。闭式胸外按压结果为：RAPe46．4±9．5／6．3±6．0；PAPe62．9±16．7／10．0±3．8；FAPe103．6±27．0／14．6±5．9，开胸按压时：RAPo21．7±3．0／9．0±5．6；PAPo29．9±4．2／11．9±4．6；FAPo46．9±5．0／17．6±8．9；单位为mmHg。通过比较发现：1．胸外按压产生的收缩压大于开胸按压（P＜0．005），而舒张压反而低于开胸条件下之值；2．力量是维持收缩压的主要因素，而频率是维持舒张压的主要因素；3．在胸外或开胸按压下，上述三处压力不等。FAP、PAP间接代表了左室压、右室压，它们之间存在着一个承受和传导压力的梯度：即左室压＞右室压＞右房压。这一发现有利于对心脏按压力量传导机制及其条件下血液动力学改变机制的认识和理解。
In order to further study the pressure transduction mechanism of human heart pressure, in this paper, 7 critically ill patients (4 cases of traumatic brain injury, 3 cases of GCS, 3 cases of multiple trauma and average ISS of 22.3) Right atrial pressure (RAP), pulmonary arterial pressure (PAP), and femoral arterial pressure (FAP) were monitored during Swan-Ganz catheterization and invasive femoral arterial pressure monitoring during routine rescue after cardiopulmonary resuscitation. Closed chest compression results were: RAPe 46.4 ± 9.5 / 6.3 ± 6.0; PAPe 62.9 ± 16.7 / 10.0 ± 3.8; FAPe 103.6 ± 27.0 / 14. 6 ± 5.9 at thoracotomy: RAPo 21.7 ± 3.0 / 9.0 ± 5.6; PAPo 29.9 ± 4.2 / 11.9 ± 4.6; FAPo 46.9 ± 5.0 /17.6 ± 8.9; mmHg. By comparison found: 1. Thoracic pressure produced systolic blood pressure greater than the thoracotomy (P <0.005), while diastolic blood pressure but lower than the value of the thoracotomy; Strength is the main factor to maintain systolic blood pressure, and frequency is the main factor to maintain diastolic blood pressure; Under chest or chest compression, the above three pressures range. FAP, PAP indirectly represent the left ventricular pressure and right ventricular pressure, there is a gradient between the pressure and pressure they carry: left ventricular pressure> right ventricular pressure> right atrial pressure. This finding is conducive to understanding and understanding of the mechanism of cardiac compression of stress transmission and its mechanism of hemodynamic changes.