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本实验以每周一次、每次10mg的剂量,将100mg和50mg镁砂粉尘(90%以上为氧化镁)分别注入两组大鼠气管内。实验共进行了18个月,其间定期处死大鼠,进行肺脏及肺引流淋巴结的病理形态学观察。结合1例工龄34年的镁矿工的尸检材料分析,初步证明镁尘对肺脏的致纤维化作用极弱。并且通过单纯氧化镁组动物实验的对照和尸检肺内粉尘的EDXA分析证明,这种极弱的致纤维化作用并非来自其中的氧化镁本身,而是镁尘中其它成份所致。
The experiment once a week, each dose of 10mg, 100mg and 50mg magnesia dust (more than 90% of magnesium oxide) were injected into the trachea of two groups. The experiment was conducted for 18 months, during which rats were sacrificed on a regular basis to observe the pathological morphology of the lungs and the draining lymph nodes. Combined with an autopsy material analysis of a 34-year-old magnesium miners, it was tentatively proved that the effect of magnesium dust on lung fibrosis was very weak. And by simple magnesium oxide group animal experiments control and autopsy of lung dust EDXA analysis proved that this very weak fibrosis does not come from the magnesia itself, but the magnesium in other components.