论文部分内容阅读
本文报告头部外伤引起视神经萎缩15例,均为单眼。其中视力完全丧失者10例,只有光感者1例,眼前手动至0.02者4例。 视神经萎缩的原因,我们推断为视神经骨管及其附近眶骨质损伤,故均做了视神经孔后前斜位(Rhees位)摄影。仅1例视神经孔有改变,14例正常。用x线双侧视神经管及其附近眶骨正面体层摄影法,上述15例均发现异常,其中蝶骨大、小翼骨折1例,蝶骨小翼骨折9例,视神经骨管骨折5例。从检查的结果可以说明,视神经萎缩是由于视神经骨管及其附近眶骨质损伤引起直接或间接视神经损伤导致萎缩。
This article reports 15 cases of optic nerve atrophy caused by head trauma, both monocular. One case of complete loss of vision in 10 cases, only light perception in 1 case, immediate to 0.02 in 4 cases. The reason for optic atrophy, we infer that the optic nerve and the surrounding orbital bone injury, it was done after the optic nerve hole anterior oblique (Rhees bit) photography. Only 1 case of optic nerve hole has changed, 14 cases were normal. With bilateral x-ray bilateral optic canal and its adjacent orbital front body tomography, the above-mentioned 15 cases were found abnormal, including sphenoid and small wing fracture in 1 case, sphenoid small-wing fracture in 9 cases, optic nerve and bone fracture in 5 cases . From the examination results can be explained that optic atrophy is due to the optic nerve orbital bone and its vicinity of the orbital bone damage caused by direct or indirect optic nerve damage resulting in atrophy.