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创伤引起尿路结石不被人们所重视,国内外报道不多,本文报道11例,对其病因、诊断、治疗、预防等问题进行讨论。临床资料本组伤后均经 X 线证实无结石存在资料如下。讨论一、创伤后产生尿路结石的原因本组病例均有不同部位的创伤性骨折,伤后钙磷代谢变化较大,骨质的普遍脱钙,使血钙和尿钙增高;由于长期卧床,限制活动,在4～6周时,脱钙更为明显,血钙和尿钙显著增加;另外长期卧床使尿流极积易发生尿路感染,也成为促进尿石形成的局部因素之一。在发生尿路感染时,某些细菌能分解尿素释出氨,使尿碱化,有利于磷酸盐的沉积,形成所谓“感染石”.同时,由于尿素被细菌分解面减少,丧失了其保护胶体的作用,于是尿液
Trauma caused by urinary tract stones are not valued people, not many reports at home and abroad, this article reports 11 cases, the etiology, diagnosis, treatment, prevention and other issues are discussed. Clinical data of this group were confirmed by X-ray after the existence of non-stone as follows. Discussion First, the causes of post-traumatic urinary tract stones in this group of patients have traumatic fractures in different parts of the trauma changes in calcium and phosphorus metabolism, bone decalcification generally, so that blood calcium and urinary calcium increased; as a result of prolonged bed rest , 4 to 6 weeks, the decalcification is more obvious, serum calcium and urinary calcium increased significantly; in addition to prolonged bed rest urinary tract easily lead to urinary tract infection, but also to promote urolithiasis one of the local factors . In the event of urinary tract infection, some bacteria can break down urea release ammonia, so that urine alkalization, is conducive to the deposition of phosphate, the formation of the so-called “infected stone.” At the same time, due to bacterial decomposition of urea decreased, the loss of its protection Colloidal effect, so urine