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眼压的升高长久以来被认为是临床确诊青光眼的基本依据之一。正常眼压性青光眼（ＮＰＧ）的概念对眼压的诊断意义提出新的认识。目前有关正常眼压性青光眼发病机制的研究形成两种观点，即自身免疫缺陷机制和眼压调节机制。这两种机制研究的客观背景提示，正常眼压性青光眼的发病机制可能是自身免疫及眼压调节两种因素相互作用的结果。对于ＮＰＧ患者发病机制的研究将拓深对青光眼发病机制、临床诊断及治疗措施的传统认识。
IOP elevation has long been considered as one of the basic evidence of clinically diagnosed glaucoma. The concept of normal tension glaucoma (NPG) put forward a new understanding of the diagnostic significance of intraocular pressure. At present, there are two kinds of research about the pathogenesis of normal tension glaucoma, namely autoimmune defect mechanism and intraocular pressure regulating mechanism. The objective background of these two mechanisms suggests that the pathogenesis of normal-tension glaucoma may be the result of the interaction of both autoimmune and intraocular pressure (IOP) changes. Research on the pathogenesis of NPG patients will deepen the traditional understanding of the pathogenesis, clinical diagnosis and treatment of glaucoma.