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采用模拟再灌的缺氧－复氧模型，在细胞水平直接观察到缺氧－复氧可致心肌细胞产生一氧化氮明显增加，并诱导心肌细胞凋亡；首次发现“缺氧后处理”可减轻心肌复氧损伤，明显提高心肌细胞存活率（Ｐ＜ ０．０５）。为更有效救治心肌缺血提供了新的依据
Hypoxia-reoxygenation model was used to simulate reperfusion. Hypoxia-reoxygenation was observed directly at the cellular level, and the production of nitric oxide was significantly increased in cardiomyocytes and apoptosis of cardiomyocytes was also observed. For the first time, “hypoxic postconditioning” Reduce myocardial reoxygenation injury, significantly improve the survival rate of myocardial cells (P <0.05). It provides a new basis for more effective treatment of myocardial ischemia