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急性心肌梗塞(AMI)时,心电图(ECG)ST段压低产生的机制说法不一,尤其在下壁Q波性AMJ初期,心前ST段压低的机制更是众说纷云。以往把ST段抬高看作为产生Q波性AMI的必具条件,而把ST段压低(有或无继发T波倒置)看作为心内膜下缺血的标志。近来几项研究表明:ST段抬高是产生Q波性或非Q波性AMI的决定因素,非Q波性AMI初期ST段抬高竟达三分之二。非Q波性AMI发生机理:梗塞初期,相关的冠状动脉发生血栓
Acute myocardial infarction (AMI), the electrocardiogram (ECG) ST segment depression have different mechanisms, especially in the inferior Q wave AMJ early, ST-segment depression mechanism is even more numerous. In the past, ST-segment elevation was considered a necessary condition for the development of Q-wave AMI, and ST-segment depression (with or without secondary T-wave inversion) was considered a sign of subendocardial ischemia. Several recent studies have shown that ST-segment elevation is the determining factor for the development of Q-wave or non-Q-wave AMI, and ST-segment elevation in non-Q-wave AMI is as high as two-thirds. Non-Q wave AMI mechanism of occurrence: the initial infarction, the occurrence of coronary thrombosis