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作者观察了5例全部肾脏灌注不良伴有血浆肾素活性低下的病人,并作了分析。例1患慢性问质性肾炎,肾功能衰竭,作右肾摘除及肾移植。术后移植肾动脉狭窄。例2动脉硬化所致双侧肾动脉狭窄。例3患遗传性肾炎,双侧肾摘除及一侧肾移植,术后肾动脉狭窄。例4慢性复发性泌尿道感染,左主肾动脉发育不良。例5第五胸椎水平主动脉缩窄。5例在未限制盐摄入时,血浆肾素正常或低下,血压均显著升高。 4例中有3例在限盐和用速尿使血浆容量缩减后,血浆肾素活性显著升高。但却不能影响动脉血压。在人和动物实验性单侧肾缺血所致高血压时,受
The authors looked at all 5 patients with poor renal perfusion and plasma renin activity and analyzed them. Example 1 suffering from chronic interstitial nephritis, renal failure, removal of the right kidney and kidney transplantation. Postoperative renal artery stenosis. Example 2 Arteriosclerosis caused by bilateral renal artery stenosis. Example 3 suffering from hereditary nephritis, bilateral renal removal and kidney transplantation, renal artery stenosis. Example 4 chronic recurrent urinary tract infection, left main renal artery dysplasia. Example 5 Fifth thoracic horizontal aortic constriction. 5 cases of salt intake is not limited, the normal or low plasma renin, blood pressure were significantly increased. In 4 of 3 cases, plasma renin activity was significantly increased after salt restriction and furosemide reduction of plasma volume. But it can not affect arterial blood pressure. In humans and animals due to experimental unilateral renal ischemia caused by hypertension