应用利血平耗竭体内儿茶酚胺和肾上腺切除法制造急性心肌梗塞（ＡＭＩ）模型，发现ＡＭＩ后出现的血粘度升高，红细胞变形能力下降，红细胞压积升高，血浆粘度升高及红细胞聚集性增强等血液流变学变化是交感－肾上腺髓质系统兴奋所致。其中红细胞压积增加的主要原因是交感神经兴奋，而其它改变可能为交感神经肾上腺髓质共同作用的结果 Application of reserpine depleted catecholamine and adrenalectomy in the manufacture of acute myocardial infarction (AMI) model and found that after AMI increased blood viscosity, reduced erythrocyte deformability, elevated hematocrit, increased plasma viscosity and increased erythrocyte aggregation Hemorheological changes such as sympathetic - adrenal medulla system excited. One of the main reasons for the increase in hematocrit is sympathetic activation, while other changes may be the result of synergy of the adrenal medulla