慢性肺源性心脏病(简称肺心病)的基本病理变化是由于肺动脉高压导致右心功能和结构的异常改变直至心力衰竭。而肺动脉高压形成的机制除功能性肺血管痉挛和解剖性肺血管床减少外,近年来的研究还证明因缺氧、酸中毒和感染等原因造成血粘度增加、血液高凝,这也是使肺动脉压增高的重要因素。笔者于 The basic pathological changes of chronic cor pulmonale (referred to as pulmonary heart disease) are due to abnormal changes in right heart function and structure due to pulmonary hypertension until heart failure. The mechanism of pulmonary hypertension in addition to functional pulmonary vasospasm and anatomic pulmonary vascular bed reduction in recent years, studies have also shown that hypoxia, acidosis and infection caused by increased blood viscosity, hypercoagulability, which is the pulmonary artery The pressure increases the important factor. The author at