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曼氏血吸虫病人的肾脏损害从肾小球结缔组织细胞增生到混合的增生性和膜性肾小球肾炎都已经有过报告。免疫荧光和电子显微镜研究又发现在肾小球毛细血管壁上有IgG、IgM、补体C_3和电子致密沉着物。多年来认为肾脏损害的主要发病机理是大量可溶性抗原抗体复合物流入肾脏而引起的。近来已在实验感染日本血吸虫和曼氏血吸虫的动物以及一个肝脾型曼氏血吸虫病并发肾病接受肾脏移植的病人的肾小球内,经免疫荧光法检出了血吸虫抗原。作者检查7例肝脾型曼氏血吸虫病死者
Renal damage in patients with Schistosoma mansoni has been reported from proliferative glomerular connective tissue cells to mixed proliferative and membranous glomerulonephritis. Immunofluorescence and electron microscopy also found IgG, IgM, complement C_3 and electron dense deposits on the glomerular capillaries. Over the years that the main pathogenesis of kidney damage is a large number of soluble antigen-antibody complexes caused by the flow of the kidneys. Schistosoma antigens have recently been detected by immunofluorescence in animals experimentally infected with Schistosoma japonicum and Schistosoma mansoni as well as in the glomerulus of a patient with a liver-spleen-type Schistosoma japonicum complicated with nephropathy undergoing kidney transplantation. The authors examined seven cases of liver and spleen Schistosoma mansoni dead