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急性肾小球肾炎患者出现短暂性高血钾已见报道,但其发生机制尚不完全清楚。作者晚近对此进行了探讨。 4例男性感染后肾小球肾炎患者,血清钾5.7～6.7mmol/L。均有急性肾小球肾炎的临床表现(水肿、高血压、蛋白尿、血尿及抗链“O”升高),其中3例接受了肾活检并证实了诊断,该4例与血清钾正常的18例对照,其血浆醛固酮及肾素活性显著降
Acute glomerulonephritis in patients with transient hyperkalemia has been reported, but the mechanism is not yet fully understood. The author recently explored this. 4 cases of male patients with glomerulonephritis, serum potassium 5.7 ~ 6.7mmol / L. All had clinical manifestations of acute glomerulonephritis (edema, hypertension, proteinuria, hematuria and anti-chain “O” elevation), of which 3 received renal biopsy and confirmed the diagnosis of the 4 patients with normal serum potassium 18 cases of control, the plasma aldosterone and renin activity decreased significantly