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我们自1973年以来曾用克山病病区与非病区出产的粮食对大鼠进行多次饲养观察。结果均表明,病区饲料组检出心肌实质性病变,生长迟缓,平均存活日数少,心脏重量相对增大,胸腺及褐脂重量较低,对一些牵累因子耐受性差,机体对某些微量元素示踪代谢的吸收率较高。病区饲料组大鼠与非病区饲料组大鼠之间存在明显差异。实验证明,病区粮中存在致动物心肌坏死和影响生长因子。为了进一步探讨病因,我们拟对此种差异的发病机理首先进行研究。鉴于近来大量资料证明cAMP 是机体组织内的一种重要活性物
Since 1973, we have used Keshan disease ward and non-ward production of food on rats for many feeding observation. The results showed that, the ward feed group detected myocardial parenchymal lesions, growth retardation, the average number of days of survival less, the relative increase in heart weight, thymus and brown fat weight is low, to some of the factors involved in poor tolerance, the body of some trace Absorption of elemental tracer metabolism is higher. Ward feed group rats and non-ward feed group there is a significant difference between. Experiments show that the existence of animal ward myocardial necrosis and affect growth factors. In order to further explore the cause, we intend to study the pathogenesis of such differences first. In view of the recent large amount of data to prove that cAMP is an important active substance in body tissues