关于乙型肝炎病毒与肝癌之间关系的流行病学证据的一种可能的解释,最近由巴黎巴斯德研究所的科研人员提出。他们认为,病毒可能整合到宿主的处于不活动状态的癌基因(可能引起癌肿的基因)附近的遗传物质内。在癌基因附近发生的病毒的整合作用,使得癌基因激活和癌的突发。 A possible explanation for the epidemiological evidence of the relationship between hepatitis B virus and liver cancer was recently proposed by researchers at the Pasteur Institute in Paris. In their opinion, the virus may be integrated into genetic material in the vicinity of the host’s inactive oncogene, a gene that may cause cancer. The integration of the virus that occurs in the vicinity of the oncogene makes oncogene activation and the onset of cancer.