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通过２４例脑梗塞病人和２１例正常对照外周血淋巴细胞进行体外培养，检测其ＭＢＰ和ＭＰＬＰ反应性Ｔ淋巴细胞所分泌的ＩＦＮ－γ，结果发现实验组ＭＢＰ和ＭＰＬＰ反应性ＩＦＮ－γ的产生均高于对照组，差异显著（Ｐ值分别小于０．００２５和０．０２５），提示脑梗塞发生后缺血所致的脑组织的破坏使髓鞘成分作为隐蔽抗原释放，导致相应的髓鞘蛋白反应性Ｔ淋巴细胞的增殖，这些Ｔ细胞及其所分泌的细胞因子可能是脑缺血后造成脑组织损害的第二位因素。
24 cases of cerebral infarction patients and 21 cases of normal control peripheral blood lymphocytes were cultured in vitro to detect MBP and MPLP reactive T lymphocytes secreted IFN-γ and found that the experimental group MBP and MPLP-reactive IFN-γ production Were higher than the control group, the difference was significant (P values were less than 0.0025 and 0.025, respectively), suggesting that cerebral infarction caused by ischemia after the brain tissue damage to the myelin component as a hidden antigen release, resulting in the corresponding myelin The proliferation of protein-reactive T lymphocytes, these T cells and their secreted cytokines may be brain damage caused by cerebral ischemia second factor.