桥脑中央髓鞘溶解症(central portime myclinolysis CPM)病因及发病机理与血清Na~+浓度异常密切相关。现将我们收治1例报告如下: 患者,男性,71岁,因左眼闭合不全36小时,右下肢无力24小时入院。既往有高血压病史10年,饮酒史10年,每日饮酒量为250克,但已戒酒2年,查体:营养中等,心、肺、腹均未见异常。神经系统检查:神志清楚,左侧周围性面神经瘫痪,发音不清,饮水呛咳,吞咽困难,双侧软腭上提尚好,咽反射存在,伸舌向右歪斜,右下肢肌力Ⅳ级,右下肢腱反射较左侧活跃,右侧Babinski征(+),辅助检查:血常规、大、小便常规、肝、肾功能、血脂均正常,电解质、血 The etiology and pathogenesis of central portomy myclinolysis (CPM) are closely related to the abnormality of serum Na ~ + concentration. Now we receive a report as follows: The patient, male, aged 71, due to incomplete left eye 36 hours, right lower limb weakness 24 hours admission. Previous history of hypertension 10 years, 10 years of drinking history, daily drinking amount of 250 grams, but has abstained for 2 years, physical examination: medium nutrition, heart, lung, abdominal were no abnormalities. Nervous system examination: conscious, the left side of facial nerve paralysis, unclear pronunciation, drinking water, cough, difficulty swallowing, both sides of the soft palate is good, pharyngeal reflex present, tongue tilt to the right, the right lower extremity muscle strength Ⅳ, right Lower extremity tendon reflex than the left active, right Babinski sign (+), auxiliary examination: blood, large, urinary routine, liver and kidney function, blood lipids are normal, electrolytes, blood