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100例慢性苯中毒的骨髓象中,轻度增生减低81例,中度增生减低4例,轻度增生明显活跃15例;其中41例有轻重不等的细胞成熟障碍以及中毒性形态异常如毒性颗粒与空泡等。各例如骨髓细胞分类都见到中性分叶粒细胞增加,从正常的10%增加到20～30%;结合周围血液中中性粒细胞计数长期减少,表明慢性苯中毒的骨髓除了造血功能异常外,还有释放障碍。这些情况与一般再生障碍性贫血相似,但慢性苯中毒的骨髓变化较轻。
100 cases of chronic benzene poisoning bone marrow like, 81 cases of mild hyperplasia, moderate hyperplasia in 4 cases, mild hyperplasia was significantly active in 15 cases; of which 41 cases of varying degrees of maturity of cells and the toxicity of toxic morphological abnormalities such as toxicity Particles and vacuoles and so on. For example, the classification of bone marrow cells showed increased neutrophil granulocytes, from 10% of normal to 20-30%; combined with peripheral blood neutrophil count decreased over time, indicating that chronic benzene poisoning in addition to bone marrow abnormalities in hematopoietic function In addition, there are obstacles to release. These conditions are similar to the general aplastic anemia, but there is less bone marrow changes in chronic benzene poisoning.