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应用细胞因子,细菌内毒素和甲醛固定的细菌刺激小鼠腹腔巨噬细胞观察其诱生一氧化氮(NO)的能力,结果发现LPS,IFN-γ+IL-2能够诱生NO,IFN-γ和高浓度的M-CSF与LPS具有协同作用。细菌和BCG也具有诱生NO的能力。还观察了巨噬细胞诱生NO的动力学变化。
The ability of inducing nitric oxide (NO) in mice peritoneal macrophages was stimulated by cytokines, bacterial endotoxins and formalin-fixed bacteria. LPS, IFN-γ and IL-2 were able to induce NO and IFN- Gamma and high concentrations of M-CSF have synergistic effects with LPS. Bacteria and BCG also have the ability to induce NO. The kinetic changes of NO induced by macrophages were also observed.