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因汞中毒可以损害脑、肝、肾,而肝脏中汞的浓度常达脑的两倍或两倍以上。作者研究用检查肝损害的血清转氨酶水平的升高是否能作为汞中毒时神经症状出现前的一项指标;并研究了硒和胱氨酸对汞中毒的防护作用对这种酶的影响。实验一:216只年龄两周的鹌鹑,分为六组,分别喂以含0、0.5、2、8、20和32 ppm汞(氯化甲基汞,下同)的半合成饲料。间隔4天,每组杀死6只动物,取血分析SGPT及SGOT。吃含汞饲料的
Mercury poisoning can damage the brain, liver and kidney, and the mercury in the liver is often twice or more than that of the brain. The authors investigated whether elevated serum aminotransferases for detecting liver damage could be used as an indicator of neurological symptoms before mercury poisoning; and the effect of selenium and cystine on the protection of mercury poisoning. Experiment 1: Two hundred and sixty-two quails were divided into six groups and fed semi-synthetic feed containing 0, 0.5, 2, 8, 20 and 32 ppm mercury (methylmercury chloride, the same below). At intervals of 4 days, 6 animals were killed in each group, and blood was analyzed for SGPT and SGOT. Eat mercury-containing feed