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为探讨急性脑梗塞时患者体内 脂质过氧化及纤溶状况，以指导临床诊疗。用硫代巴比妥酸比色法和酶联免疫吸附双抗体夹心法分别测定了患者丙二醛（ Ｍ Ｄ Ａ） 及 Ｄ二聚体（ Ｄｄｉｍ ｅｒ） 水平，结果在急性脑梗塞时 Ｍ Ｄ Ａ 为（７１ ±１ ．４）ｎｍ ｏｌ／ Ｌ，较正常人（６ ．０±１ ．３）ｎ ｍｏｌ／ Ｌ 明显地升高（ Ｐ＜ ０ ．０１） ， Ｄｄｉｍｅｒ 在急性脑梗塞时为（１ ．７２ ±０ ．９８） ｍ ｇ／ Ｌ 较正常人（０ ．２６ ±０ ．１３） ｍｇ／ Ｌ 也明显地升高（ Ｐ＜ ０ ．０１） 。提示脑梗塞时机体内存在脂质过氧化及纤溶活性的增强，适当予以抗氧化及溶栓治疗是降低病人死亡率，提高治愈率的一项有效措施。
In order to explore the patients with acute cerebral infarction lipid peroxidation and fibrinolysis, to guide the clinical diagnosis and treatment. Thiobarbituric acid colorimetric method and enzyme-linked immunosorbent double antibody sandwich method were measured in patients with malondialdehyde (M D A) and D dimer levels (D dim er) levels in the results of acute cerebral infarction When M D A was (7.1 ± 1.4) nm ol / L, which was significantly higher than that of normal people (6.0 ± 1.3) n mol / L (P <0.01) (1.72 ± 0.98) m g / L in acute cerebral infarction was significantly higher than that in normal people (0.26 ± 0.13) mg / L (P <0.01). Tip of cerebral infarction in the presence of lipid peroxidation and fibrinolytic activity increased, appropriate anti-oxidant and thrombolytic therapy is to reduce the patient’s mortality rate and improve the cure rate is an effective measure.