典型的格林—巴利综合合征(Guillain-B-arre Syndrkme,GBS)诊断要点是病前13周有感染史,急性或亚急性起病,四肢对称性弛缓性瘫痪,脑神经损害,脑脊液蛋白—细胞分离现象。脑神经损害以双侧面瘫常见,其次是舌咽迷走神经麻痹,眼外肌,三叉神经损害少见。近收一病人恰与此相反,首先眼外肌麻痹,依次出现舌咽、迷走神经麻痹,双侧面神经损害。报告如下: 患者男,41岁。因复视4d,言语不清,声音嘶哑,吞咽困难3d,以颅神经型GBS收治。患者于4d前睡眠晨醒后,无明显诱因感到复视,次日出现声音嘶哑,言语不清,吞咽困难,饮水呛咳,进行性加重,无缓解。自发病以来,神志清,无发热,无两便障碍, The classic diagnosis of Guillain-B-arre Syndrkme (GBS) is that it has a history of infection 13 weeks before onset of illness, acute or subacute onset, flaccid symmetry of limbs, cerebral nerve damage, cerebrospinal fluid protein - Cell separation phenomenon. Cerebral nerve damage to bilateral facial paralysis is common, followed by glossopharyngeal vagal palsy, extraocular muscles, trigeminal nerve damage rare. Close to a patient just on the contrary, the first extraocular muscle paralysis, followed by glossopharyngeal vagus nerve paralysis, bilateral facial nerve damage. The report is as follows: Male patient, 41 years old. Due to diplopia 4d, unclear speech, hoarseness, difficulty swallowing 3d, with cranial nerve GBS admitted. Patients in the morning before sleep 4d wake up, no obvious incentive to double dip, the next day there was hoarse voice, unclear speech, difficulty swallowing, drinking water, cough, aggravated, no relief. Since the onset, conscious, no fever, no two obstacles,