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本文报道了芦笋（ＡｓｐａｒａｇｕｓＯｆｉｃｉｎａｌｉｓＣａｕｌｕｓ，ＦＡＣ）对ＥＡＣ的抑制作用及机理。ＦＡＣ使ＥＳＣ实体瘤荷瘤小鼠的抑瘤率达４１．４％～６４．３％；使ＥＡＣ腹水瘤小鼠生命延长率达３０．２％。投用ＦＡＣ的ＥＳＣ实体瘤荷瘤小鼠脾淋巴细胞转化刺激指数（ＳＩ）和ＮＫ细胞毒分别为对照鼠的１０．６倍及１．４倍；投用ＦＡＣ的ＥＡＣ腹水瘤荷瘤小鼠上述两个指标分别为对照鼠的２．７倍及１．３倍。１／１０ＩＤ５０ＦＰ－１、ＦＰ－３对ＥＡＣ细胞３Ｈ－脯氨酸摄取的抑制率分别为１５．３％及５．４％；１／１０ＩＤ５０ＦＰ－１、ＦＰ－３对ＥＡＣ细胞３Ｈ－ＴｄＲ摄取的抑制率分别为５９．５％及６２．０％。
This paper reports the inhibitory effect and mechanism of AsparagusOficinalis Caulus (FAC) on EAC. The tumor inhibition rate of tumor-bearing mice with ESC solid tumors by FAC was 41.4%-64.3%, and the survival rate of EAC ascites tumor mice was 30.2%. The stimulation index (SI) and NK cytotoxicity of spleen lymphocytes in tumor-bearing mice injected with FAC ESC solid tumors were 10.6-fold and 1.4-fold higher than those in control mice, respectively; EAC ascites tumor tumor-bearing mice that were administered with FAC The above two indicators were 2.7 and 1.3 times that of the control mice, respectively. The inhibition rates of 3H-proline uptake of EAC cells by 1/10 ID50FP-1 and FP-3 were 15.3% and 5.4%, respectively; 1/10 ID50FP-1 and FP-3 uptake of 3H-TdR in EAC cells. The inhibition rates were 59.5% and 62.0%, respectively.