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急性一氧化碳中毒主要是经呼吸道吸入肺内的一氧化碳与血红蛋白很快结合成碳氧血红蛋白(COHb),由于COHb无携氧能力,致使组织发生严重缺氧,出现中枢神经系统、呼吸系统和循环系统等中毒症状,中毒过深者当场死亡,重者如救治不当则成为持续植物人状态或遗留偏瘫、痴呆等后遗症。本研究旨在探讨纳洛酮(NLX)对急性重度一氧化碳中毒的临床疗效。1 材料与方法1.1 临床资料本文收集我院急诊科和兰州军区乌鲁木齐总医院急诊科1994年～1999年急性重度一氧化碳中毒病例共536例。NLX组303例,男186例,女117例,年龄1～70岁,平
Acute carbon monoxide poisoning is mainly through the respiratory tract inhaled carbon monoxide and hemoglobin quickly combined into carboxyhemoglobin (COHb), due to COHb no oxygen carrying capacity, resulting in severe hypoxia, the emergence of the central nervous system, respiratory and circulatory system Symptoms of poisoning, poisoning were killed on the spot, severe cases such as improper treatment has become a persistent vegetative state or legacy hemiplegia, dementia and other sequelae. This study aimed to investigate the clinical efficacy of naloxone (NLX) in acute severe carbon monoxide poisoning. 1 Materials and Methods 1.1 Clinical Data This article collected emergency department of our hospital and Urumqi General Hospital of Lanzhou Military Region from 1994 to 1999 a total of 536 cases of acute severe carbon monoxide poisoning. There were 303 cases in NLX group, including 186 males and 117 females, aged from 1 to 70 years old