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例1,男,67岁。因患高心病、心衰Ⅱ。于1986年11月2日入院。入院时血压190/100mmHg,尿常规正常,CO_2CP23.2mmol/L,BUN5.4mmol/L,血肌酐106.1umol/L。胸透:靴型心,心搏减弱,主动脉舒展。ECG:左室肥大劳累。入院后口服双氢克尿噻50mg,每日3次,心痛定10mg口服,每日3次,以及给氧护心治疗。3天后血压150/90mmHg,心衰基本控制,继用双氢克尿噻10天后病人逐渐出现少尿,尿比重1.010,血钾7.34mmol/L,BUN18.7mmol/L,血肌酐329.64umol/L。ECG为窦室传导。考虑为双氢克尿噻所致
Example 1, male, 67 years old. Because of high heart disease, heart failure Ⅱ. On November 2, 1986 admission. Blood pressure 190 / 100mmHg on admission, normal urinalysis, CO 2 CP 23.2mmol / L, BUN 5.4mmol / L, serum creatinine 106.1umol / L. Chest through: Boots heart, weakened heart, aortic stretch. ECG: Left ventricular hypertrophy. Hydrochlorothiazide 50mg after oral administration, 3 times a day, 10mg nifedipine orally, 3 times a day, and oxygen therapy. 3 days later, the blood pressure was 150/90 mmHg, and the heart failure was basically controlled. After the patients received hydrochlorothiazide ten days later, the patients gradually developed oliguria, urine specific gravity 1.010, serum potassium 7.34mmol / L, BUN18.7mmol / L, serum creatinine 329.64umol / L . ECG is sinus room conduction. Considered to be caused by hydrochlorothiazide