论文部分内容阅读
脑缺血诱导神经细胞表达的热休克蛋白 70 (ＨＳＰ70 ) ,被认为在脑缺血引起的损伤中具有脑保护作用。西比灵选择性阻断Ｃａ2 + 过度内流 ,减轻缺血性神经损伤。本文用肾血管性高血压大鼠 (ＲＨＲ)制作脑缺血再灌注模型 ,评价西比灵对脑缺血再灌注损伤的保护作用并探讨其脑保
Cerebral ischemia induces the expression of heat shock protein 70 (HSP70) in nerve cells and is thought to have a neuroprotective effect in the damage induced by cerebral ischemia. Seibeline selectively blocks excessive Ca2 + influx, reduce ischemic nerve injury. In this paper, we used Renal Vascular Hypertensive Rats (RHR) to make cerebral ischemia-reperfusion model to evaluate the protective effect of Sibelium on cerebral ischemia-reperfusion injury