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突触前和突触后细胞内钙离子 ([Ca2 +]i)在短时程和长时程突触的可塑性中 ,发挥着重要的信息传递作用。兴奋后残留 [Ca2 +]i,可以激发短时程突触增强。突触前 [Ca2 +]i 可以影响被抑制的突触前膜囊泡的更新 ,并准确编码突触前和突触后信息 ,产生截然相反的长时程突触修饰 (LTP或LTD)。
The presynaptic and post-synaptic intracellular calcium ([Ca2 +] i) plays an important message-passing role in the short-term and long-term synaptic plasticity. After excitement residual [Ca2 +] i, can stimulate short-term synaptic enhancement. Presynaptic [Ca2 +] i can affect the repression of the inhibited presynaptic vesicle and accurately encode presynaptic and postsynaptic messages, producing opposite long-term synaptic modifications (LTP or LTD).