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急性心肌梗死(AMI)的发生,90%是由冠脉内粥样斑块破裂血栓形成引起[1]。而血栓形成与纤溶系统的活性改变密切相关,其中组织型纤溶酶原激活剂(t-PA)及其抑制物(PAI-1)对调节纤溶活性起着决定性的作用[2]。为此,本研究对18例AMI患者的纤溶活
Acute myocardial infarction (AMI) occurs in 90% of cases caused by atherosclerotic plaque rupture thrombosis [1]. Thrombosis is closely related to changes of fibrinolytic system activity. Tissue plasminogen activator (t-PA) and its inhibitor (PAI-1) play a decisive role in the regulation of fibrinolytic activity [2]. To this end, the study of 18 patients with AMI fibrinolytic activity