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有研究表明,砷可以引起机体内砷致机体脂质过氧化的产生及抗氧化能力降低,从而造成机体的氧化损伤〔1-2〕。而肝脏是砷主要累及的靶组织〔3〕,但砷对肝细胞的作用特点研究较少。研究提示,活性氧(ROS)和线粒体膜电位(ΔΨm)在细胞凋亡中发挥着重要作用〔4-5〕。为了进一步探讨砷
Studies have shown that arsenic can cause the body caused by arsenic-induced lipid peroxidation and antioxidant capacity decreased, resulting in the body’s oxidative damage 〔1-2〕. The liver is the main target tissue involved arsenic 〔3〕, but arsenic on the role of hepatocytes less studied. Research suggests that reactive oxygen species (ROS) and mitochondrial membrane potential (ΔΨm) play an important role in apoptosis [4-5]. In order to further explore arsenic