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对体质性生长迟缓和家族性身材矮小儿童的发锌、血锌含量及其中部分口服甲基睾丸酮儿童服药前后的发锌、血锌含量分别给予测定,所得结果表明: 一、男性生殖器官发育达第三阶段以后,其锌含量显著高于第一、第二阶段。二、血清睾丸酮浓度(至250ng/dl止)与锌含量呈现直线相关关系。三、服用甲基睾丸酮后锌含量增加,体质性生长迟缓儿童的增加尤为明显。由此可见,内源性产生的睾丸酮含量增加及外源性供给睾丸酮均伴有锌含量的增加。故推测:体质性生长迟缓儿童的睾丸酮和促性腺激素相对不足可引起锌含量的减少,而锌含量的减少反过来又会进而推迟第二性征的出现和使生长更为迟缓。
On constitutional growth retardation and familial short stature of children hair zinc, blood zinc content and some of oral methyl testosterone children before and after treatment of hair zinc, blood zinc levels were given to measure the results showed that: First, the male reproductive organs up to After the third stage, its zinc content was significantly higher than the first and second stages. Second, serum testosterone concentration (to 250ng / dl only) and the zinc content showed a linear correlation. Third, after taking methyl testosterone increased zinc content, physical growth retardation in children is particularly evident. Thus, endogenous production of testosterone and exogenous supply of testosterone are accompanied by increased zinc content. Therefore, speculation: constitutional retardation in children with relatively insufficient testosterone and gonadotropin can cause a decrease in zinc content, and zinc reduction in turn will further delay the emergence of secondary sexual characteristics and to make growth more slowly.