坏死性炎症最终可导致肝硬化,表现为肝脏广泛的间质纤维化、小叶结构改变及假小叶形成。肝内静脉血栓常出现于肝硬化患者,并与肝硬化程度相关,而肝内小静脉和肝血窦闭塞是导致肝脏结构改变的潜在促进因素。各种血栓危险因子在血栓形成中的机制已经阐明,但是它们在慢性病毒性肝炎或肝硬化中的作用尚无研究报道,本研究对慢性乙型和丙型肝炎患者的一些原发与继发性血栓形成因素进行了流行 Necrotic inflammation can eventually lead to cirrhosis, manifested as extensive hepatic interstitial fibrosis, lobular structure changes and pseudolobule formation. Intrahepatic venous thrombosis often occurs in patients with cirrhosis, and with the degree of cirrhosis, and intrahepatic venous and hepatic sinusoids occlusion is a potential contributing factor to the structural changes of the liver. The mechanisms of thrombosis risk factors in thrombosis have been elucidated, but their role in chronic viral hepatitis or cirrhosis has not been reported. In this study, some primary and secondary patients with chronic hepatitis B and C Thrombotic factors have been prevalent