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本实验观察益母草治疗心肌缺血再灌注损伤过程中氧自由基的变化。结果显示，益母草能明显抑制血中和心肌组织中的丙二醛（MDA）的产生，保护超氧化物歧化酶（SOD）和谷胱甘肽过氧化物酶（GSH－PX）的活性（与对照组相比P＜0．05～0．01）。表明益母草治疗心肌缺血再灌注损伤的机制可能与益母草减轻氧自由基对心肌的损害有关。
This experiment was conducted to observe the changes of oxygen free radicals in the treatment of myocardial ischemia-reperfusion injury by Herba Leonuri. The results showed that Motherwort could significantly inhibit the production of malondialdehyde (MDA) in blood and myocardial tissues, and protect the activity of superoxide dismutase (SOD) and glutathione peroxidase (GSH-PX). P<0.05-0.01 compared to the control group. It was suggested that the mechanism of Yimucao treating myocardial ischemia-reperfusion injury may be related to the decrease of myocardial damage induced by Leonurus.