肺水肿是指肺的血管外部分,包括肺间质和肺泡间隙中水的异常积聚.这是多种发病机理引起的急性呼吸功能不足时的早期表现,造成肺顺应性减少和氧交换损害,导致动脉血氧过低症.过去,肺水肿是心力衰竭的同义词,提示肺毛细管流体静力压过高.近来发现,在监护室内见到的多数肺水肿,尤其是外科病人,其原因为非心源性的,而显然由于肺毛细血管渗透性增加所致.关于肺水肿仍有许多问题未曾解决.作者一直致力于发展对非心源性水肿的治疗.本文旨在探索肺水肿形成的机制和各种因素对它的影响.肺液体运动的基本问题 Pulmonary edema refers to the extravascular portion of the lung, including the abnormal accumulation of water in the interstitial and alveolar spaces of the lung, an early manifestation of a variety of pathogenesis-induced acute respiratory insufficiency, resulting in reduced lung compliance and oxygen exchange damage, Leading to arterial hypoxemia. In the past, pulmonary edema is synonymous with heart failure, suggesting that pulmonary capillary hydrostatic pressure too much recently found in the custody room to see the majority of pulmonary edema, especially in surgical patients, the reason is not Cardiogenic, and apparently due to an increase in pulmonary capillary permeability.About the pulmonary edema, there are still many problems have not been solved.And the authors have been committed to the development of non-cardiac edema.This paper aims to explore the mechanism of pulmonary edema And various factors on its impact on the basic issue of pulmonary fluid movement