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玉米大斑病菌主要通过附着胞的机械穿透力侵染寄主,三环唑能够降低玉米大斑病菌对寄主的致病力。对三环唑培养下玉米大斑病菌附着胞形成、膨压变化过程以及病菌的侵染效能等进行分析,探讨三环唑抑制玉米大斑病菌侵染的机制。结果表明,5μg/m L三环唑药剂培养下的玉米大斑病菌在附着胞发育时期和附着胞成熟期的膨压较不加药剂处理分别下降了0.22 MPa和1.72 MPa,接菌后72 h对寄主的侵染效率也明显下降,三环唑主要通过影响成熟期附着胞形成高膨压降低病菌的侵染能力。
Rhizoctonia solani infects hosts mainly through the mechanical penetration of attachment cells, and tricyclazole can reduce the virulence of S. turcica to the host. The formation of appressorium, the process of turgor pressure and the infection efficiency of S. turcica were analyzed in tricyclazole culture to investigate the mechanism of tricyclazole in inhibiting S. turcica infection. The results showed that turpentinera turcica cultured in 5μg / mL tricyclazole had a decrease of 0.22 MPa and 1.72 MPa in turgor pressure and attachment stage compared with no treatment, respectively. After 72 h of inoculation, Infection efficiency to the host also decreased obviously. Tricyclazole reduced the infection ability of the bacteria mainly by affecting the high turgor of the appressorium in the mature stage.