纽约大学的科研人员发现,动脉粥样硬化斑块细胞中含有使细胞生长失控的基因,当这些遗传成分转移到其他细胞时,促使这些细胞成为肿瘤样的东西。尽管动粥样硬化很常见,但在心脏病学方面,其发病原因仍不清楚,西雅图华盛顿大学分别由科研人员发展了两个理论学说。一个是损伤应答假说,这种假说的论据是动脉粥样硬化斑块出现在血管被损伤的地方;另一个是单克隆假说,即斑块主要是由单种平滑肌细胞所产生的良性肿瘤。纽约大学在10月号国立科学协会会议录上的报告中提供了单克隆假说的机制。阿瑟佩恩等通过一种专门检测基因的仪器设备,发现了动脉粥样硬化平滑肌细胞 Researchers at New York University found that atherosclerotic plaque cells contain genes that cause cells to become out of control, and when those genetic elements are transferred to other cells, they cause the cells to become tumor-like. Although atherothrombosis is common, the etiology of etiology is still unclear, and two theoretical doctrines have been developed by researchers at the University of Washington in Seattle. One is the damage response hypothesis, which argues for the presence of atherosclerotic plaque in areas where blood vessels are damaged; the other is the monoclonal hypothesis that the plaque is a benign tumor that is primarily produced by a single type of smooth muscle cell. New York University provided a mechanism for the monoclonal hypotheses in a report to the National Science Council meeting in October. Arthur Payne and other instruments through a special test of the gene found atherosclerosis smooth muscle cells