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本文用放射免疫法测定39例正常人及58例慢性肾脏疾病患者的血浆PC含量。1.慢性肾炎普通型7例,PC平均值为2.657±0.44 mg/L,与正常对照3.24±0.4 mg/L比明显降低(P<0.05);2.肾病综合征6例,PC含量为6.446±2.504 mg/L,与对照组比明显升高(P<0.05);3.氮质血症组8例,PC平均值为3.424±0.363 mg/L,与正常对照比无明显差异(P>0.05);4.尿毒症非透析组26例,PC平均值为4.902±2.035 mg/L与正常比明显升高(P<0.001);5.尿毒症透析组,其中血透6例PC平均值为2.63±0.547 mg/L,与正常对照比明显下降(P<0.05),腹透组5例PC值为5.56±1.952 mg/L,与正常对照比明显升高(P<0.05),在慢性肾脏疾病中PC降低可有血栓形成促使肾病恶化,PC增高可有抗凝作用引起出血倾向,在肾病综合征中PC增高有助于对抗高凝状态。
In this paper, radioimmunoassay 39 cases of normal and 58 cases of chronic kidney disease in patients with plasma PC content. 1. The average type of chronic nephritis in 7 cases was 2.657 ± 0.44 mg / L, which was significantly lower than that of normal control (3.24 ± 0.4 mg / L) (P <0.05). 2. Nephrotic syndrome in 6 cases and PC content was 6.446 ± 2.504 mg / L, which was significantly higher than that of the control group (P <0.05) .3. In the azotemia group, the average value of PC was 3.424 ± 0.363 mg / L in 8 patients, 0.05) .4. Uremic non-dialysis group 26 cases, PC average 4.902 ± 2.035 mg / L and normal ratio was significantly higher (P <0.001); 5 uremia dialysis group, including hemodialysis in 6 cases of PC average (2.63 ± 0.547 mg / L, P <0.05). The PC value of 5 cases in PD group was 5.56 ± 1.952 mg / L, which was significantly higher than the normal control group (P <0.05) Decreased PC in renal disease may lead to thrombosis worsened renal disease, increased PC may have bleeding tendency caused by anticoagulant, PC in nephrotic syndrome increased resistance to hypercoagulable state.