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目的:探讨汉族、维吾尔族婴儿肝炎综合征患儿的病因构成比并比较二者之间是否存在差异。方法:对确诊婴儿肝炎综合征的维吾尔族患儿107例及汉族患儿121例病因分析并进行比较。结果:病原学检查结果,维吾尔族:巨细胞包涵体病毒感染为48例(44.9%),乙型肝炎病毒22例(20.6%),EB病毒10例(9.3%),丙型肝炎4例(3.7%),HIV感染2例(1.8%),梅毒螺旋体感染2例(1.8%),细菌培养阳性6例(5.6%),先天性胆道闭锁3例(2.8%),其他10例(15.0%)。汉族:巨细胞包涵体病毒感染为66例(54.5%),乙型肝炎病毒17例(14.0%),EB病毒6例(5.0%),丙型肝炎例(3.3%),HIV感染1例(0.8%),梅毒螺旋体感染1例(0.8%),细菌培养阳性4例(3.3%),先天性胆道闭锁2例(1.7%),其他21例(19.8%)。结论:婴儿肝炎综合征病因构成比在维吾尔族及汉族患儿之间无统计学差异,均为巨细胞包涵体病毒感染占第一位,乙肝病毒感染占第二位。
Objective: To investigate the etiopathogenisis of infants with Hepatitis Syndrome in Han and Uygur ethnic Chinese and to compare the differences between the two. Methods: The etiology of 107 cases of Uighur children diagnosed with infant hepatitis syndrome and Han children were analyzed and compared. Results: The etiology of Uygur: 48 cases (44.9%) of viral infection of cytomegalovirus, 22 cases of hepatitis B virus (20.6%), 10 cases of Epstein-Barr virus (9.3%) and 4 cases of hepatitis C 3.7%), HIV infection in 2 cases (1.8%), Treponema pallidum infection in 2 cases (1.8%), bacterial culture positive in 6 cases (5.6%), congenital biliary atresia in 3 cases (2.8% ). Han nationality: Cytomegalovirus infection was found in 66 cases (54.5%), hepatitis B virus in 17 cases (14.0%), Epstein-Barr virus in 6 cases (5.0%), hepatitis C case 0.8%), Treponema pallidum infection in 1 case (0.8%), bacterial culture positive in 4 cases (3.3%), congenital biliary atresia in 2 cases (1.7%) and the other 21 cases (19.8%). Conclusion: The etiological factor of infantile hepatitis syndrome is not statistically different between Uygur and Han children, both of which are giant cell inclusion virus infection and hepatitis B virus infection second.