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背景血液制品中存在的白细胞被证实能促成许多包括供体抗受体和受体抗供体免疫反应的发生。美喹他嗪PRT处理使病原体减少是基于暴露核黄素和光照后,可使其核酸复制失活,有证据显示该处理也可致有核白细胞功能失活。研究设计与方法从接受美喹他嗪治疗或对经十四酰佛波乙酸酯
Background The presence of white blood cells in blood products has been shown to contribute to many occurrences of donor immune responses including donor anti-receptors and receptors. Mequitazine PRT treatment reduces pathogens based on exposure to riboflavin and light, inactivating nucleic acid replication, and evidence suggests that this treatment may also cause deactivation of nucleated leukocytes. Study design and methods from receiving mequitazine treatment or via tetradecanoylphosphates acetate