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对Bell氏面瘫病损的部位仍有争议。有人认为是面神经颞骨段水肿。但动物实验表明,舌下神经核破坏,可产生远端神经干的肿胀。已有研究发现,Bell氏面瘫的脑脊液中,髓磷脂变性产物增多,听觉脑干反应异常。提示中枢神经系统(CNS)受累及有些Bell面瘫是多灶性疾患。为进一步弄清该病神经系统受累的范围,作者对28名急性单侧Bell氏面瘫的病人进行了临床调查。结果发现:急性期仅有7人表现为单独的面神经功能障碍。其余21人皆有不同的神经系统广泛受累的症状。最常见的(20/28人,71%)是患侧或健侧(2人)三叉神经功能障碍,表现为该区域感觉减退或过敏,及角膜反射障碍。3人有舌咽神经受累,
The site of Bell’s facial paralysis remains controversial. Some people think that facial nerve segment temporal bone edema. However, animal experiments show that the destruction of the hypoglossal nucleus can produce swelling of the distal nerve trunk. It has been found that Bell’s facial paralysis cerebrospinal fluid, myelin degeneration products increased, auditory brainstem response abnormalities. Suggesting that central nervous system (CNS) involvement and some Bell facial paralysis are multifocal disorders. To further understand the extent of the disease involved in the nervous system, the authors of 28 patients with acute Bell’s facial paralysis in a clinical investigation. The results showed that: in the acute phase, only 7 people showed single facial nerve dysfunction. The remaining 21 people all had different symptoms of extensive involvement of the nervous system. The most common (20/28, 71%) were ipsilateral or contralateral (2) trigeminal dysfunction characterized by hypoalgesia or allergy in this area and corneal reflexes. 3 people have glossopharyngeal nerve involvement,