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急性重度一氧化碳中毒所致的精神障碍伴肝功能损害,临床上并非罕见,但同时并发糖代谢紊乱的病例,尚不多见。我院曾收治一例,现报告如下。病例介绍张某,男,32岁。患者因紧闭门窗,煤火取暖,入睡12小时后才被发现,四肢抽搐,当时已昏迷,而于1984年1月24日急诊入院。既往体健,从无精神异常及多饮、多食、多尿的表现。体检:T38.4℃,P122次,R34次,BP12/8kPa。颜面潮红,口唇樱红色,深度昏迷,呼吸急促,四肢阵发性抽动,瞳孔中等散大,对光反射消失,角膜反射消失。呼吸节律不整,两肺闻及湿性罗音,心率122次,肝脾未扪及。双侧巴彬斯奇氏反应阳性。化验:1月24日,肝功能
Mental disorders caused by acute severe carbon monoxide poisoning with liver damage, clinical is not uncommon, but at the same time complicated with complications of glucose metabolism are rare. Our hospital has admitted a case, the report is as follows now. Case introduction Zhang, male, 32 years old. Patients closed the doors and windows, fire coal, 12 hours after falling asleep was found, limbs convulsions, was unconscious, and in January 24, 1984 emergency admission. Past physical health, from no mental disorders and drink more, eat more, more urine performance. Physical examination: T38.4 ℃, P122 times, R34 times, BP12 / 8kPa. Face flush, lips fuchsia, deep coma, shortness of breath, paroxysmal twitching limbs, middle pupil dilated, light reflex disappeared, corneal reflex disappeared. Respiratory irregular rhythm, lungs and wet rales smell, heart rate 122 times, liver and spleen not palpable. Babinski’s reaction was positive on both sides. Laboratory: January 24, liver function