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通过对5例肾移植后并发急性呼吸窘迫综合征（ARDS）病人的氧动力学监测，结果发现所有病。可能存在高氧耗型氧缺陷。它的机制是由于感染通过细胞团子介导系统性炎性反应综合征（SIRS）。治疗的重点应该集中在寻找病因学证据以便控制SIRS和降低氧消耗。其次，应该采取各种措施维持一个超正常值水平的氧输送。
Five patients with acute respiratory distress syndrome (ARDS) after the renal transplantation were monitored for oxygenation and all the disease was found. There may be oxygen-deficient oxygen defects. Its mechanism is due to infection mediated through the cell mass of systemic inflammatory response syndrome (SIRS). Treatment should focus on finding etiological evidence to control SIRS and reduce oxygen consumption. Second, various measures should be taken to maintain an ultra-normal level of oxygen delivery.