以前认为血管紧张素Ⅱ（ＡｎｇⅡ）阻滞剂、限制蛋白摄人是通过降低肾小球毛细血管压从而减少肾组织损伤，延缓肾脏疾病进展，最近的资料表明这两种方法也可能不通过降低血压，而是通过其它机制延缓疾病进展，即限制修复过程，这种修复过程如果没有自身限制将会导致组织纤维化 Angiotensin II (AngⅡ) blockers were previously considered to limit protein ingestion by reducing glomerular capillary pressure and thus reducing renal tissue damage and slowing the progression of kidney disease. Recent data suggest that these two approaches may not be reduced by lowering Blood pressure, but through other mechanisms to delay disease progression, which limits the repair process, this repair process without its own limitations will lead to tissue fibrosis