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患者男,40岁,1990年3月15日酒后骑车摔伤头面部、昏迷数十分钟,当地医院诊为脑震荡,右眼球后出血.1个月后行右眼球后注射.α—糜蛋白酶2000u,不见好转,眼球突出逐渐加重,眼睑和球结膜水肿,伴同侧偏头痛.于1990年5月15日来本院就诊,以眼眶内占位性病变收入院.检查:视力:右0.5,左1.5;眼球突出度:右21mm,左13mm,眶距115mm;右眼:眼睑高度肿胀;球结膜下部高度水肿,突出于睑裂外,球结膜血管纡曲、怒张,呈暗红色;在眼睑部和颞侧眶部可闻及响亮且与脉搏一致的吹风样杂音;角膜知觉明显减退;瞳孔散大,直径约为5mm,光反射迟钝;视乳头色泽淡,黄斑中心凹反射存在,视网膜静脉轻度纡曲、扩张,周边部视网膜反光增强,未见出血和渗出;上睑下垂;眼球各方向运动受限,呈现固视状态,额部皮肤感觉减退,
Male, 40 years old, March 15, 1990 drunk riding a wounded head and face, coma for several minutes, the local hospital diagnosed as concussion, bleeding after the right eye .1 months after the right eye after injection .α- Chymotrypsin 2000u, did not improve, the eyeballs gradually increased, eyelid and conjunctival edema, with ipsilateral migraine headache .In May 15, 1990 to our hospital, orbital space-occupying lesions income hospital .Check: visual acuity: Right 0.5, left 1.5; eyeball protrusion: right 21mm, left 13mm, orbital distance 115mm; right eye: eyelid height swelling; bulbar conjunctiva height edema, prominent in the palpebral fissure, bulbar conjunctiva vascular curvature, ; In the eyelid and temporal orbital Department can smell and loud and consistent with the pulse of the hair-like murmur; corneal sensation was significantly diminished; mydriasis, diameter of about 5mm, slow light reflex; optic disc color faded foveal reflex exists , Mild retinal vein curvature, expansion, the peripheral retinal reflex enhanced, no bleeding and exudation; blepharops; eye movement restricted in all directions, showing the fixation state, forehead skin sensory decline,