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1965年及1966年即有人报告听神经瘤病人的外淋巴中含有高浓度蛋白(正常人的5～15倍)。1971年Silverstein曾用免疫电泳及免疫扩散法测验,谓蛋白含量增加为血清蛋白蓄积干外淋巴所致。认为听神经瘤妨碍内耳血管的生理功能,以致血清蛋白漏于外淋巴中。继肿瘤压迫所致的静脉滞留或非重要原因,因迷路静脉汇流于蜗导水管的静脉,并不通过内耳道。外淋巴中蛋白含最增加可用以诊断较小的听神经瘤(X线片未见内耳道扩大者)。本文作者提出以下问题:在听神经瘤患者中,外淋巴中是否含有其他蛋白(非血清蛋白)非免
In 1965 and 1966, it was reported that patients with acoustic neuromas had high concentrations of proteins in the perilymph (5 to 15 times normal). In 1971, Silverstein used an immunoelectrophoresis and immunodiffusion method to test that the protein content increased as the accumulation of serum protein in the dry perilymph. It is believed that acoustic neuromas interfere with the physiological function of the blood vessels of the inner ear, so that the serum protein leaks into the perilymph. Following venous retention or non-significant causes due to tumor compression, the labyrinth vein confluences in the vein of the worm-guided waterway and does not pass through the inner ear canal. The most increase in protein content in the perilymph can be used to diagnose smaller acoustic neuromas (enhancement of the inner ear canal without X-ray). The authors ask the following questions: In patients with acoustic neuromas, whether the perilymph contains other proteins (non-serum proteins) is not immune