Gene damages of mitochondrial DNA encoding cytochrome oxidase of intestinal epithelial cells in hemo

来源 :Chinese Journal of Traumatology | 被引量 : 0次 | 上传用户:redbird_zdc
下载到本地 , 更方便阅读
声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架
论文部分内容阅读
Objective: To investigate the detrimental effects of hemorrhagic shock on the structure and function of mitochondria DNA (mtDNA) encoding cytochrome oxidase genes in intestinal epithelial cells. Methods: Wistar rats were used and divided into two groups: hemorrhagic shock group and control group. Hemorrhagic shock model of rats was utilized in this experiment. The mtDNA was extracted from the intestinal epithelial cells and amplified by polymerase chain reaction (PCR) with different primers of cytochrome oxidase (COX I, COX II and COX III). The products of PCR were directly sequenced. Results: Hemorrhagic shock could result in the point mutagenesis in mitochondrial genome encoding cytochrome oxidase (COX I and COX II). There were 4, 4, 22, 16, 35 point mutations in COX I from 5545 to 6838 bp in 5 shocked rats. There were five point mutations in COX II from 7191 to 7542 bp at the site of t7191c, t7212c, a7386g, a7483g, c7542g in 1 shocked rat. There was no mutation found in COX III. Conclusions: Hemorrhagic shock could significantly induce the damage of the gene of cytochrome oxidase encoded by mtDNA. Objective: To investigate the detrimental effects of hemorrhagic shock on the structure and function of mitochondria DNA (mtDNA) encoding cytochrome oxidase genes in intestinal epithelial cells. Methods: Wistar rats were used and divided into two groups: hemorrhagic shock group and control group. Hemorrhagic shock model of rats was taken in this experiment. The mtDNA was extracted from the intestinal epithelial cells and amplified by polymerase chain reaction (PCR) with different primers of cytochrome oxidase (COX I, COX II and COX III). The products of PCR were directly sequenced. Results: Hemorrhagic shock could result in the point mutagenesis in mitochondrial genome encoding cytochrome oxidase (COX I and COX II). There were 4, 4, 22, 16, 35 point mutations in COX I from 5545 to 6838 bp in 5 There were five point mutations in COX II from 7191 to 7542 bp at the site of t7191c, t7212c, a7386g, a7483g, c7542g in 1 shocked rat. There was no mutation found in COX III. Con clusions: Hemorrhagic shock could precipit induce the damage of the gene of cytochrome oxidase encoded by mtDNA.
其他文献
新课标中明确指出:学生是语文学习的主人。语文教学应激发学生的学习兴趣,注重培养学生自主学习的意识和习惯,鼓励学生选择适合自己的学习方式。因此教师应创造性地理解和使
第一届全军医学情报专业组、情报网会议在总后卫生部的领导和关怀下,于1986年5月7~11日在重庆第三军医大学召开。参加会议的有各军医、军种的总医院、军医学校和军事医学研究
请下载后查看,本文暂不支持在线获取查看简介。 Please download to view, this article does not support online access to view profile.
期刊
为了保证战伤伤员饮水的需要,我们配制试用了口服补液粉(简称R.O.S口服补液),并在紧张激烈艰苦的战斗中,在伤员中试用了口服补液,使伤员受伤后及时补充含电解质的水份,维持
本文报道6例肝豆状核变性的患儿,病程从15天至5年不等,其起病类型有:神经系统症状者2例,肝炎者1例,溶血性贫血者1例,无症状者2例。有1例的临床经过与暂时性血管内溶血伴急性
本文报导了11例慢性肾功能衰竭患者由于长期血液透析而发生的进行性透析性脑病(Progressivedialytic encephalopathy)。这病与急性透析性脑病和肾功能衰竭的末期表现不同。1
湘江北去,橘子洲头。喜看2015年,教育改革热浪滔滔,我们飞扬梦想,收获荣光。2015年,国家教育改革顺利推进。城乡义务教育经费保障机制不断完善、中小学教师职称制度改革进一
请下载后查看,本文暂不支持在线获取查看简介。 Please download to view, this article does not support online access to view profile.
在苏州逗留的那段时间,我结识了一个小男孩。他有一张清俊得如姣女般的脸庞,一双透着灵气的大眼睛又让你读出男孩的“调皮”来,小巧玲珑惹人喜爱。和他接触中,我发现了他的一
本文报导了小儿烟雾病临床预后的研究结果。全组共24例,其中男性5例,女性19例,发病年龄在一岁以下的2例,平均年龄为5岁4个月。除了5例以外,其余的随访观察时间都在4年以上。