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过量一氧化碳经呼吸道入血后 ,立即与血红蛋白结合成碳氧血红蛋白 ,引起血液性缺氧而造成的脑损害称为一氧化碳中毒性脑病。有的患者在急性一氧化碳中毒后 ,从急性中毒昏迷中恢复 ,经过数天至二个月的表现正常或接近正常的间歇期 ,而后又出现以急性痴呆状态为特征的全脑性症状
Excessive carbon monoxide through the respiratory tract into the blood, immediately with hemoglobin into carboxyhemoglobin, causing blood hypoxia caused by brain damage called carbon monoxide poisoning encephalopathy. Some patients recover from acute poisoning coma after acute carbon monoxide poisoning and after several days to two months they perform normal or near-normal intermittent periods and then develop whole-brain symptoms characterized by acute dementia