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5.2 反应性氧族清除一氧化氮在许多血管病理中,一氧化氮(NO)合成率的变化加上 NO 清除的增加,会导致 NO 生物利用度的降低,引起 NO 信号的减少,在一些病例中还会出现 NO 源性的毒性。NO 生物利用度的减少最基本的机制是O_2~(?)对 NO 的作用。正常生理条件下,O_2~(?)在超氧化物歧化酶(SOD)的作用下去氧化转化为过氧化氢
5.2 Reactive Oxygen Removal of Nitric Oxide In many vascular pathologies, changes in the rate of nitric oxide (NO) synthesis, combined with an increase in NO clearance, leads to a decrease in NO bioavailability resulting in a reduction in NO signaling. In some cases There will be NO-derived toxicity. The most basic mechanism of NO bioavailability reduction is O 2 ~ (?) On NO. O_2 ~ (?) Under normal physiological conditions, the role of superoxide dismutase (SOD) into the oxidation of hydrogen peroxide