论文部分内容阅读
小白鼠接触二氧化氮后肺内出现蛋白漏出液,大概是由于上皮和内皮细胞损伤引起细胞脱落和蛋白漏入肺泡、支气管树。从接触污染空气引起有关体液因素变化的观点来看,在肺以外可能也有毛细管的损伤。鉴于肾脏可“正常地”从尿中排出少量蛋白,因而可把它用以揭示毛细管损伤和/或小管坏死引起蛋白丢失的重要指示器。本研究应用盘式凝胶电泳、化学试验以及比重测定法,评价豚鼠接触0.5和0.4ppm 二氧化氮对尿蛋白含量的影响。
After exposure to nitrogen dioxide mouse protein leakage occurs in the lungs, probably due to epithelial and endothelial cell damage caused by cell loss and protein leakage into the alveoli, bronchial tree. From the point of view of exposure to contaminated air, changes in body fluid may also have capillary damage beyond the lungs. Since the kidney can “expel” a small amount of protein “normally” from the urine, it can be used to reveal an important indicator of protein damage caused by capillary damage and / or tubular necrosis. In this study, the effects of exposure to 0.5 and 0.4ppm of nitric oxide on urinary protein content in guinea pigs were evaluated by disk gel electrophoresis, chemical tests and specific gravity measurement.