急性肺损伤(ALI)和急性呼吸窘迫综合征(ARDS)是各种原因引起的肺实质弥漫性损害,以急性发展的双肺毛玻璃改变为特征[1]。ALI/ARDS与脓毒症的发病机制相似,均涉及失控的宿主防御反应,其病理变化包括凝血系统增强、纤溶系统抑制。新的证据显示,恢复凝血纤溶系统平衡可能是ALI/ARD Acute lung injury (ALI) and acute respiratory distress syndrome (ARDS) are diffuse lesions of the lung that are caused by a variety of causes and are characterized by an acute development of double-glass frosted glass [1]. ALI / ARDS resembles the pathogenesis of sepsis and involves uncontrolled host defense responses. Pathological changes include increased coagulation and fibrinolysis. New evidence suggests that restoring the balance of coagulation and fibrinolysis may be ALI / ARD