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氯磷定所致凝血机制障碍国内尚未见报道.最近,我们遇一例报告如下:患者女,18岁,因间歇性鼻出血12天,皮肤瘀斑一周;于83年7月20日入院.病人于半月前晚9时许服敌敌畏50毫升,约30分钟后被家人发现其口吐白沫,不省人事,即送当地医院经洗胃,用阿托品,氯磷定(1克每日2次加入液体中静点,连用三天)治疗,于次晨5时苏醒.10天前(即停氯磷定的第2天)无明显诱因突然鼻衄,量约500毫升,予以凡士林纱布堵塞、止血药物、出血方停止.一周前又发作一次,用纱布压迫,
Clofibrate due to clotting mechanism obstacles have not been reported in the country.Recently, we encounter a case of the following reports: The patient female, 18 years old, due to intermittent epistaxis for 12 days, skin ecchymosis week; July 83, 83 admitted to the patient. At half past nine on the evening of 9 o’clock to take DDVP 50 ml, about 30 minutes after the discovery of the family members of their mouth foaming, unconsciousness, namely to the local hospital after gastric lavage, with atropine, chlorophosphonates (1 g twice daily Added to the liquid in the static point, once every three days) treatment, wake up at 5 am the next morning .10 days ago (that is, the first two days of phosphorus trichloride) no significant incentive sudden epistaxis, the amount of about 500 ml, vaseline gauze blocked, Hemostatic drugs stop bleeding a week before the attack once again with gauze oppression,