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植物免疫受体FLS2能够感受植物病原菌鞭毛蛋白N端的22氨基酸(flg22)并迅速激活植物的免疫反应。BIK1作为一种细胞质类受体激酶(RLCK)和FLS2形成免疫受体复合体共同调控着植物免疫受体的激活。植物NADPH氧化酶RbohD对植物受到flg22诱导之后产生ROS和气孔关闭是必须的。但是有关RbohD的调节机制并不清楚。中国科学院遗传与发育生物学研究所科研人员近期通过生化和生理学实验揭示,BIK1正调控flg22诱导的钙离子细胞内流,RbohD与免疫受体形成复合体参与免疫反应的调节,BIK1和RbohD直接相互作用并且flg22可以诱导RbohD发生磷酸化并且从免疫受体复合体上解离,进一步研
The plant immune receptor FLS2 is capable of sensing 22 amino acids (flg22) at the N-terminal end of the plant pathogenic flagellin and rapidly activating plant immune responses. BIK1 as a cytoplasmic receptor kinase (RLCK) and FLS2 form an immune receptor complex that together regulate the activation of plant immune receptors. The plant NADPH oxidase RbohD is essential for plants to produce ROS and stomatal closure after induction by flg22. However, the regulatory mechanism of RbohD is not clear. Researchers at the Institute of Genetics and Developmental Biology, Chinese Academy of Sciences recently revealed through biochemical and physiological experiments that BIK1 regulates flg22-induced influx of intracellular Ca2 +. RbohD and immune receptors form a complex that regulates the immune response. BIK1 and RbohD interact directly with each other And flg22 can induce RbohD to be phosphorylated and dissociated from the immunoreceptor complex, and further research